You run your hand along your dog’s back and notice white flakes on the couch cushion, or you see your dog scratching at dry, dull skin more often than usual. Dry skin is one of the most common reasons owners consult a veterinarian about their dog’s coat, and it ranges from a simple fix (add fish oil, increase humidity) to an early sign of hypothyroidism or parasites that need prompt treatment. This guide covers all the clinically relevant causes with the detail needed to work effectively with your veterinarian. Healthy canine skin has three functional layers relevant to dryness. The epidermis is the outermost layer; its outermost portion, the stratum corneum, consists of flattened dead keratinocytes embedded in a lipid matrix of ceramides, free fatty acids, and cholesterol that acts as a water-impermeable barrier. Below the epidermis lies the dermis, which contains collagen, blood vessels, nerve endings, and the hair follicles. The hypodermis (subcutaneous fat) provides insulation and energy storage. The sebaceous glands, located in the dermis adjacent to each hair follicle, secrete sebum: a mixture of lipids, cholesterol, and wax esters that coats the hair shaft, lubricates the skin surface, and contributes to the barrier function. When sebaceous secretion is reduced (hypothyroidism, dehydration, extreme low humidity), when the lipid matrix is depleted (fatty acid deficiency, over-bathing with harsh shampoos), or when the keratinization process is abnormal (primary seborrhea), the stratum corneum loses its water-retention capacity and transepidermal water loss increases, producing the clinical signs of dry skin. Cold outdoor air holds less water vapor than warm air. When this cold air enters a heated home and is warmed by the furnace or baseboard heaters, the relative humidity drops dramatically, often to 15-25% in cold climates during winter. This dry air draws moisture from the skin surface faster than the sebaceous glands can replenish it. The result is seasonal dry skin that worsens in winter, improves in summer, and affects both the dog and the human members of the household. A humidifier maintaining indoor relative humidity at 40-60% significantly reduces this seasonal dry skin. Dogs housed in air-conditioned spaces in summer can experience a similar effect from the dehumidifying action of air conditioning. The skin barrier’s lipid matrix is built from dietary fatty acids. Linoleic acid (an omega-6 fatty acid from plant oils) is an essential fatty acid in dogs; it is the precursor to ceramides and prostaglandins that maintain stratum corneum integrity. Deficiency of linoleic acid produces scaling, hair loss, and poor coat quality. Omega-3 fatty acids (EPA and DHA from fish oil) reduce skin inflammation and support barrier function. Dogs on generic or very low-quality diets may be deficient in one or both; even premium diets can become deficient if stored for too long (fatty acids oxidize over time), stored improperly (heat and light accelerate oxidation), or if the dog has fat malabsorption from a GI condition. Zinc deficiency causes a specific syndrome (zinc-responsive dermatosis) with crusting and scaling around the face, muzzle, and footpads, particularly in Arctic breeds (Siberian Huskies, Alaskan Malamutes) that have a genetic predisposition to zinc malabsorption, and in rapidly growing large-breed puppies fed phytate-rich grain-based diets that bind zinc. Treatment is zinc supplementation (zinc sulfate or zinc methionine 2-3 mg/kg daily) alongside dietary correction. Bathing removes sebum from the skin surface. Frequent bathing (more than once weekly for most dogs) strips the sebum layer faster than the sebaceous glands can replace it, leaving the skin temporarily dry and the hair coat dull. Human shampoos are formulated for the more acidic pH of human skin (4.5-5.5) and are too acidic for the more neutral canine skin surface (pH 6.2-7.4), disrupting the skin microbiome and barrier integrity. Dogs should be bathed with veterinary-formulated shampoos at a pH of 6.0-7.5, and no more than every 2-4 weeks for most breeds unless a medical skin condition requires more frequent therapeutic shampooing. Moisturizing conditioners applied after shampooing and left on for several minutes before rinsing help restore the lipid layer. The thyroid gland produces thyroxine (T4) and triiodothyronine (T3), hormones that regulate metabolic rate throughout the body including the skin. Hypothyroidism (insufficient thyroid hormone production) is one of the most common endocrine diseases in dogs, affecting middle-aged to older dogs of medium to large breeds including Golden Retrievers, Labrador Retrievers, Doberman Pinschers, Irish Setters, Boxers, and Cocker Spaniels. Skin changes from hypothyroidism include dry, dull, brittle hair that epilates (falls out) easily without pruritus (itching), symmetrical hair loss on the trunk and tail base (rat tail), hyperpigmentation of the skin, scaling, and myxedema (puffy thickening of the facial skin, particularly over the forehead, giving a “tragic” expression). Concurrent systemic signs include weight gain without increased appetite, lethargy, cold intolerance, bradycardia, and a slow heart rate. Diagnosis: total T4 below 1.0 mcg/dL with compatible clinical signs. Treatment: levothyroxine (Soloxine) 0.02 mg/kg twice daily; recheck total T4 four to eight weeks after starting treatment aiming for a post-pill 4-6 hour level of 2.5-3.5 mcg/dL. Skin and coat improvement typically visible at 6-12 weeks, full resolution at 3-6 months. Excess cortisol from Cushing’s disease (either a pituitary adenoma driving the adrenals or a primary adrenal tumor) causes a distinctive skin syndrome: thin, fragile, easily bruised skin with calcium deposits (calcinosis cutis), bilateral symmetrical hair loss on the trunk, a “pot-bellied” abdomen from fat redistribution, scaling, and comedones (blackheads). The skin appears dry, thin, and inelastic. Concurrent signs include polydipsia, polyuria, polyphagia, and a pot-bellied silhouette. Diagnosis requires low-dose dexamethasone suppression test (LDDST) or ACTH stimulation test. Pituitary-dependent Cushing’s is treated with trilostane (Vetoryl 2.2-6.7 mg/kg once daily) or mitotane; adrenal tumors often require surgery. Skin improvement follows successful cortisol control. Cheyletiella yasguri (dogs), Cheyletiella blakei (cats), and Cheyletiella parasitovorax (rabbits) are large surface-dwelling mites that cause heavy white scaling on the dorsal trunk (back and neck). The mites are visible to the naked eye as moving white specks beneath and among the scales, earning the name “walking dandruff.” Pruritus ranges from none to moderate. The mites are highly contagious between animals and transiently zoonotic (they cause itchy papules on human arms and torso). Any dog with sudden-onset heavy dorsal scaling should be evaluated for Cheyletiella. Diagnosis: acetate tape preparation from the scale, coat brushings onto dark paper, or skin scraping microscopy. Treatment: selamectin (Revolution) monthly for 3 doses, or an isoxazoline (Bravecto, NexGard, Simparica). All in-contact animals (including cats and rabbits) must be treated. The environment should be vacuumed thoroughly as mites can survive off-host for a few days. Atopic dogs have a genetic defect in the skin barrier: reduced ceramide production in the stratum corneum increases transepidermal water loss and allows allergen penetration that drives pruritic inflammation. Dry, flaky skin with itching in an atopic dog is therefore both a cause and an effect of the allergy: the compromised barrier allows more allergen in, worsening inflammation, which further damages the barrier. Management requires addressing both the pruritus (Apoquel, Cytopoint, immunotherapy) and actively restoring the skin barrier with ceramide-containing topical products and omega-3 supplementation. Food allergy can cause similar dry skin signs; see the food elimination trial discussion above. Primary seborrhea is a hereditary defect in keratinization that causes abnormal stratum corneum formation and sebaceous gland secretion. It presents in two forms: seborrhea sicca (dry, flaky scaling) and seborrhea oleosa (greasy, adherent scales with a rancid odor), though many dogs have mixed features. Breed predispositions include American Cocker Spaniel (most severely affected; often have seborrhea oleosa), West Highland White Terrier, Basset Hound, Irish Setter, Labrador Retriever, Doberman Pinscher, and German Shepherd. Signs appear in young dogs (under 2 years) and are progressive. Secondary Malassezia and Staphylococcus infections are almost always present and worsen the scaling and odor. Management is lifelong: medicated shampoos (selenium sulfide 1%, coal tar and sulfur combinations, salicylic acid 2%, zinc pyrithione 1%) used 2-3 times weekly; omega-3 supplementation; and in severe Cocker Spaniels, oral synthetic retinoids (isotretinoin 1-2 mg/kg once daily). There is no cure. Insufficient water intake reduces circulating blood volume and interstitial fluid available to the skin. Mild chronic dehydration produces dry, inelastic skin (a skin tent that is slow to return: gently pinch the skin over the neck or shoulder and release; in a hydrated dog it snaps back immediately; in a dehydrated dog it stays tented for one or more seconds). Dogs that eat only dry kibble with limited water access, dogs with chronic kidney disease causing increased urinary water loss, or dogs in hot environments may be mildly chronically dehydrated. Ensuring fresh water is always available, adding water to dry food, or transitioning to wet food increases daily water intake and improves skin hydration. Human moisturizers containing fragrances, propylene glycol, or alcohol can worsen skin irritation and are toxic if licked. Baby oil (mineral oil) applied to the coat is a folk remedy that leaves a greasy residue without addressing the underlying dryness, and it occludes pores. Coconut oil applied topically to the coat is popular on social media but has limited evidence and can worsen seborrhea oleosa in dogs with primary seborrhea by adding excessive lipid to already-oily scales. If using any topical product on a dog that will lick the area (paws, flanks), confirm the product is safe for ingestion at the quantities involved. Coconut oil applied to the coat is the best treatment for dry dog skin. Coconut oil contains primarily medium-chain saturated fatty acids (lauric acid, myristic acid) that are not the fatty acids the canine skin barrier requires. The stratum corneum lipid matrix depends on linoleic acid (an omega-6 fatty acid) and ceramides, neither of which coconut oil provides in meaningful amounts. Applied topically, coconut oil sits on the hair surface and pads without restoring the skin’s lipid matrix. In dogs with seborrhea oleosa, adding an oily substance to already-greasy skin worsens the condition. The evidence-based approach to dry skin from fatty acid deficiency is oral omega-3 and omega-6 supplementation, which delivers the fatty acids where they are needed: to the keratinocytes that build the skin barrier from the inside. Frequent bathing helps dry skin by cleaning away flakes. For simple environmental or nutritional dry skin, frequent bathing makes the condition worse, not better. Bathing strips the sebum layer from the skin surface, and the sebaceous glands require 24-48 hours to replenish it. Bathing more than once weekly maintains a state of chronic sebum depletion, worsening dryness. The exception is therapeutic medicated bathing prescribed for primary seborrhea or secondary infections, where the dermatologic benefit of the medicated shampoo outweighs the drying effect, and a moisturizing conditioner is used after each medicated bath to partially restore the lipid layer. For healthy dogs with dry skin, reducing bathing to once monthly and using a moisturizing dog shampoo is usually more beneficial than increasing it. Dry skin in dogs is always a seasonal, environmental problem that clears up on its own in summer. True seasonal dry skin from winter heating does improve in summer when humidity rises. However, dry skin from hypothyroidism, primary seborrhea, Cheyletiella mites, Cushing’s disease, or nutritional deficiencies is present year-round and does not resolve seasonally. A dog whose dry skin improves somewhat in summer but never fully resolves, or whose skin is dry year-round, has a non-seasonal cause that requires investigation. Any dog with dry skin not improving by the end of summer (when environmental humidity and temperature are optimal) should be evaluated by a veterinarian for an underlying medical cause. The two most evidence-based home measures for mild nutritional or environmental dry skin are: (1) adding fish oil to the diet (providing EPA + DHA at 20-55 mg/kg/day; roughly 1 teaspoon of salmon oil for a 30 lb dog) to restore the skin’s lipid barrier from the inside; and (2) running a cool-mist humidifier in rooms where the dog spends time during winter to maintain indoor humidity at 40-60%. These two measures resolve most mild cases of simple dry skin within 8-12 weeks. If dry skin is severe, accompanied by hair loss, or does not improve after two months of these measures, veterinary evaluation is needed to identify an underlying cause like hypothyroidism or mite infestation. Dandruff in dogs (white flakes on the coat and falling on furniture) is most commonly caused by: low indoor humidity in winter, insufficient dietary fatty acids, infrequent brushing that allows dead skin cells to accumulate, primary seborrhea in predisposed breeds, Cheyletiella mites (which look like moving dandruff flakes), or hypothyroidism in adult and senior dogs. Mild, seasonal dandruff in an otherwise healthy dog is usually addressed with omega-3 supplementation, a humidifier, and regular brushing. Heavy, sudden-onset dandruff on the back and neck warrants a veterinary visit to rule out Cheyletiella before trying any topical treatments, since mites require specific antiparasitic treatment to resolve. Most human lotions and moisturizers should not be applied to dogs. They are formulated for human skin pH (4.5-5.5), which is more acidic than dog skin (pH 6.2-7.4), and they often contain fragrances, alcohol, propylene glycol, or xylitol (found in some products) that are irritating or toxic to dogs. The main concern is ingestion: a dog will lick anything applied to accessible skin, and even small amounts of some human lotion ingredients can cause GI upset or toxicity. Veterinary-formulated moisturizers, ceramide sprays, and paw balms designed for dogs (such as Douxo S3, Vetoquinol Epimove, or Musher’s Secret for paws) are safe alternatives that are formulated for the correct pH and safe if licked. The standard veterinary dosing recommendation for EPA + DHA combined for skin support is 20-55 mg/kg/day. For practical guidance: a 20 lb (9 kg) dog needs approximately 180-500 mg of combined EPA + DHA daily; a 50 lb (23 kg) dog needs approximately 450-1,250 mg; a 80 lb (36 kg) dog needs approximately 720-2,000 mg. Check the fish oil product label for the combined EPA + DHA content per capsule or per teaspoon, not the total fish oil content (which includes other non-active lipids). Give fish oil with food to minimize GI upset. Start at the lower end of the range for the first 2 weeks. Visible coat improvement takes 8-12 weeks as the skin cell turnover cycle replaces old cells with new ones built from the supplemented fatty acids. Dry skin can be a sign of allergic skin disease (atopic dermatitis), but it is not exclusively an allergy sign. The key distinction is the presence and intensity of itching: atopic dogs itch significantly (paw licking, face rubbing, scratching at ears and belly), and the dry skin is part of a broader allergic syndrome. Simple nutritional or environmental dry skin with minimal itching is not allergy. If your dog has dry skin primarily during allergy seasons (spring, fall) and also licks its paws, rubs its face, or has recurrent ear infections, allergic skin disease is the likely primary cause and warrants veterinary allergy evaluation. If the dry skin is year-round without significant itching and is accompanied by weight gain and lethargy, hypothyroidism is more likely. Simple dry skin from nutritional deficiency, low humidity, hypothyroidism, or primary seborrhea is not contagious. However, Cheyletiella mites (“walking dandruff”) are highly contagious between dogs, cats, and rabbits in the household, and transiently zoonotic to humans (causing itchy red papules on the arms and torso of people who handle infested pets). If multiple pets in your home or humans in your family are itching simultaneously with a dog that has heavy dandruff, Cheyletiella mites are the most likely shared cause. Veterinary diagnosis of the dog followed by treatment of all in-contact animals resolves the problem in all household members. Sarcoptes scabiei (mange) is also transiently zoonotic and can cause dry, crusty skin and intense itching in dogs; it is transmitted by direct contact. The most likely cause is low indoor humidity from forced-air heating. In cold climates, outdoor air in winter holds very little water vapor; when this air is drawn into a home and heated to 68-72 degrees Fahrenheit, the relative humidity drops to 15-25%, well below the 40-60% that supports healthy skin. This dry air draws moisture from your dog’s skin surface faster than the sebaceous glands can replace it. The same mechanism causes dry skin in humans during winter. A cool-mist humidifier running in the dog’s main living areas is the simplest and most direct solution. If humidity management helps but does not fully resolve the seasonal dryness, omega-3 supplementation provides additional skin barrier support throughout the year. For more guides on keeping your dog healthy, browse all our Dog Health articles.
Dog Dry Skin: Causes, Home Remedies, and Treatment
Dermatology and Internal Medicine
This article is reviewed for clinical accuracy. Consult your veterinarian when dry skin is severe, persistent, or accompanied by hair loss, crusting, or systemic signs.
Key Takeaways
How Dog Skin Works: The Skin Barrier
Causes of Dry Skin in Dogs
Low Environmental Humidity and Winter Dryness
Nutritional Deficiencies (Fatty Acids and Zinc)
Over-Bathing and Harsh Shampoos
Hypothyroidism
Hyperadrenocorticism (Cushing’s Disease)
Cheyletiella (Walking Dandruff)
Atopic Dermatitis and Allergies
Primary Seborrhea (Keratinization Defects)
Dehydration
Red Flags: When Dry Skin Requires Veterinary Evaluation
Distinguishing Dry Skin from Look-Alike Conditions
Condition
Key Signs
Differentiating Feature
Environmental dry skin
Fine white flakes, dull coat, mild itch; worsens in winter
Seasonal pattern; improves with humidifier and omega-3 supplementation
Cheyletiella (“walking dandruff”)
Heavy white scales on dorsal trunk; scales appear to move
Mites visible on acetate tape prep; contagious to other pets; people in household may itch
Hypothyroidism
Dry brittle coat, symmetrical hair loss, weight gain, lethargy
Total T4 below 1.0 mcg/dL on blood panel; responds to levothyroxine
Primary seborrhea
Heavy flaking or greasy scales; young dog; breed predisposed; rancid odor
Onset under 2 years; lifelong; does not fully resolve; specific breed
Atopic dermatitis
Dry flaky skin with significant pruritus; ear, paw, and belly involvement
Itch is the dominant complaint; seasonal or year-round; responds to Apoquel or Cytopoint
Zinc-responsive dermatosis
Crusting and scaling around face, muzzle, footpads
Arctic breed or growing large-breed puppy; improves with zinc supplementation
Cushing’s disease
Thin fragile skin, calcinosis cutis, pot belly, polyuria/polydipsia
Systemic signs; abnormal ACTH stimulation test or LDDST; affects middle-aged to older dogs
Diagnostic Steps for Dog Dry Skin
Test
What It Identifies
When Used
Physical exam and history (diet, bathing frequency, environment, season, concurrent signs)
Environmental and nutritional causes; breed predisposition; concurrent systemic signs suggesting endocrine disease
Every case; often provides the diagnosis or narrows the differential significantly
Coat brushings or acetate tape prep, microscopy
Cheyletiella mites; Demodex; Malassezia yeast; bacteria
Any dog with scaling, particularly if heavy and on the dorsal trunk; quick and inexpensive
Skin scraping
Demodex canis; Sarcoptes scabiei
Young dogs with hair loss; dogs with generalized scaling and pruritus
Total T4 (thyroid hormone)
Hypothyroidism (below 1.0 mcg/dL with clinical signs)
Any adult or senior dog with dry dull coat, symmetrical hair loss, weight gain, or lethargy
CBC and serum biochemistry panel
Hyperlipidemia (hypertriglyceridemia can indicate hypothyroidism or diabetes); kidney or liver disease affecting skin secondarily; blood cell changes in endocrine disease
Dogs with dry skin that does not respond to initial management; concurrent systemic signs present
ACTH stimulation test or low-dose dexamethasone suppression test
Cushing’s disease (hyperadrenocorticism)
Dogs with thin fragile skin, calcinosis cutis, pot belly, polyuria/polydipsia
Dietary trial (elimination diet 8-12 weeks)
Food allergy contribution
Non-seasonal dry skin with concurrent pruritus not responding to environmental allergy treatment
Skin biopsy
Primary seborrhea histopathology; unusual keratinization disorders; ruling out autoimmune disease
Progressive severe scaling in a young breed-predisposed dog; or dry skin not fitting a clearer diagnosis
Treatment Options for Dog Dry Skin
Treatment
Best For
Key Details
Fish oil supplementation (EPA + DHA combined, 20-55 mg/kg/day)
Nutritional dry skin; atopic dry skin barrier support; all dogs with flaking coats
Use fish oil (salmon, sardine, anchovy), not flaxseed oil; flaxseed provides ALA which dogs convert to EPA/DHA poorly; give with food to reduce GI upset; improvement visible at 8-12 weeks as skin cells turn over; safe long-term
Dietary upgrade to omega-6 adequate food
Nutritional dry skin from linoleic acid deficiency
Look for minimum 1.5-2% dry matter linoleic acid on guaranteed analysis; store food in an airtight container in a cool, dry place to prevent oxidation; check bag freshness dates; use within 4-6 weeks of opening
Humidifier (maintain 40-60% indoor relative humidity)
Winter/seasonal environmental dry skin
Cool-mist ultrasonic humidifiers are safest with pets (no hot steam burn risk); run in rooms where the dog spends most time; clean weekly to prevent mold growth; most cost-effective first step for seasonal dry skin
Moisturizing veterinary shampoo and conditioner
Mild dry skin; maintenance after therapeutic shampooing
Look for shampoos containing colloidal oatmeal (Aveeno veterinary), ceramides, or aloe vera; pH 6.0-7.5; avoid human shampoos (wrong pH); leave conditioner on 5-10 minutes before rinsing; bathe no more than every 2-4 weeks unless medically indicated
Medicated shampoo (selenium sulfide 1%, coal tar, salicylic acid 2%, zinc pyrithione)
Primary seborrhea; Cheyletiella (when used as treatment adjunct); heavy scaling
Keratolytic shampoos loosen and remove scale; selenium sulfide and coal tar defatten oily seborrhea; salicylic acid loosens dry scale; leave in contact 10-15 minutes; use 2-3 times weekly initially, then reduce to maintenance frequency
Levothyroxine sodium (Soloxine, Thyro-Tabs) 0.02 mg/kg twice daily
Hypothyroidism confirmed by total T4 below 1.0 mcg/dL
Prescription required; recheck total T4 four to eight weeks after starting, targeting post-pill 4-6 hour level 2.5-3.5 mcg/dL; skin and coat improvement at 6-12 weeks; lifelong treatment; do not use without confirmed hypothyroidism (can cause hyperthyroidism if given to a euthyroid dog)
Selamectin (Revolution) or isoxazoline parasiticide
Cheyletiella mites
Monthly selamectin for 3 doses or a single dose of fluralaner/afoxolaner/sarolaner is highly effective; treat all in-contact pets simultaneously; vacuum environment to remove mite eggs and debris
Zinc supplementation (zinc sulfate or zinc methionine 2-3 mg/kg daily)
Zinc-responsive dermatosis in Arctic breeds or growing large-breed puppies
Give with food; improvement within 4-6 weeks; Arctic breeds often require lifelong supplementation; growing puppies may resolve after diet correction; ensure dietary zinc is adequate (at least 120 mg zinc/kg dry matter in puppy food)
Topical ceramide or lipid spray (Douxo S3 Calm, Vetoquinol Epimove)
Atopic dry skin; skin barrier restoration in any dry skin condition
Applied directly to skin between baths; restores ceramide and lipid content of stratum corneum; particularly useful for atopic dogs and any dog with a disrupted skin barrier; safe and gentle, no systemic effects
Home Care: Practical Steps for Dry Skin
Age-Specific Considerations
Puppies (Under 12 Months)
Adult Dogs (1-8 Years)
Senior Dogs (8+ Years)
Breed-Specific Risks for Dry Skin
Breed
Primary Risk
Key Notes
American Cocker Spaniel
Primary seborrhea (most severe among all breeds)
Both seborrhea sicca and seborrhea oleosa occur; secondary Malassezia infections are nearly universal; lifelong medicated shampoo regimen required; oral retinoids (isotretinoin) used in severe cases
West Highland White Terrier
Primary seborrhea; atopic dermatitis
White coat makes scaling very visible; also prone to atopy that exacerbates dry skin; Malassezia overgrowth common secondary to both conditions
Siberian Husky, Alaskan Malamute
Zinc-responsive dermatosis
Genetic predisposition to zinc malabsorption; scaling and crusting particularly prominent on face and footpads; often require lifelong zinc supplementation despite an adequate-zinc diet
Golden Retriever, Labrador Retriever
Hypothyroidism; atopic dermatitis
Both breeds are among the highest-risk for hypothyroidism; coat changes are often the first owner-noticed sign before systemic signs develop; annual total T4 screening in dogs over 5 years is reasonable in these breeds
Doberman Pinscher
Hypothyroidism; color dilution alopecia
Dobermans have one of the highest rates of hypothyroidism; blue and fawn-colored Dobermans can develop color dilution alopecia (a hair follicle dysplasia) causing dry, flaky skin and patchy hair loss in the dilute-colored areas, unrelated to thyroid status
Basset Hound, Irish Setter
Primary seborrhea
Both breeds listed in breed predisposition lists for primary seborrhea; Basset Hounds also have increased interdigital skin fold infections that can contribute to localized scaling
Cost to Diagnose and Treat Dog Dry Skin in the US
Service / Treatment
Typical US Cost
Veterinary exam
$60-$150
Skin scraping and cytology
$40-$90
Total T4 thyroid test
$45-$90
Free T4 by equilibrium dialysis (more accurate thyroid test)
$80-$150
CBC and serum biochemistry panel
$120-$250
ACTH stimulation test (Cushing’s)
$200-$400
Fish oil supplement (monthly, medium dog)
$15-$35
Veterinary medicated shampoo (12 oz)
$20-$45
Levothyroxine (Soloxine, monthly, medium dog)
$20-$50
Skin biopsy
$200-$400 (including pathology)
Myths and Facts About Dog Dry Skin
Frequently Asked Questions About Dog Dry Skin
What is the best home remedy for dog dry skin?
Why does my dog have dandruff?
Can I use human lotion or moisturizer on my dog’s dry skin?
How much fish oil should I give my dog for dry skin?
Does dry dog skin mean my dog has allergies?
Is dry skin in dogs contagious to humans or other pets?
Why does my dog have dry skin in winter but not summer?
Reviewed by a Licensed Veterinary Doctor (DVM)
Products to Avoid on Dry Dog Skin
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